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Literature summary extracted from

  • Mann, N.S.; Mann, S.K.
    Enterokinase (1994), Proc. Soc. Exp. Biol. Med., 206, 114-118.
    View publication on PubMed

Natural Substrates/ Products (Substrates)

EC Number Natural Substrates Organism Comment (Nat. Sub.) Natural Products Comment (Nat. Pro.) Rev. Reac.
3.4.21.9 Trypsinogen + H2O Homo sapiens enterokinase deficiency is a distinct clinical entity characterized by diarrhea, failure to thrive, hypoproteinemia, and edema. Acquired enterokinase deficiency may occur in some diffuse small bowel diseases. Steatorhea of cellac sprue may be due partly to the fact that deficiency of secretin and cholecystokinin may interfere with the action of enterokinase ?
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 ?

Organism

EC Number Organism UniProt Comment Textmining
3.4.21.9 Homo sapiens
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Posttranslational Modification

EC Number Posttranslational Modification Comment Organism
3.4.21.9 proteolytic modification secreted as a proenzyme, enterokinase converts trypsinogen to trypsin Homo sapiens
3.4.21.9 side-chain modification glycoprotein Homo sapiens

Source Tissue

EC Number Source Tissue Comment Organism Textmining
3.4.21.9 duodenum mucosa of the first and second portion of the duodenum Homo sapiens
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3.4.21.9 jejunum jejunal mucosa Homo sapiens
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Substrates and Products (Substrate)

EC Number Substrates Comment Substrates Organism Products Comment (Products) Rev. Reac.
3.4.21.9 Trypsinogen + H2O enterokinase deficiency is a distinct clinical entity characterized by diarrhea, failure to thrive, hypoproteinemia, and edema. Acquired enterokinase deficiency may occur in some diffuse small bowel diseases. Steatorhea of cellac sprue may be due partly to the fact that deficiency of secretin and cholecystokinin may interfere with the action of enterokinase Homo sapiens ?
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 ?